[size=4][b]What are the Symptoms of Gout - Uric Acid Testing[/b][/size][hr]
[size=large][b]Definition[/b][/size][hr] Uric acid tests are tests that are done to measure the levels of uric acid in blood serum or in urine.
[size=large][b]Purpose[/b][/size][hr]
[size=medium][b]Urine test[/b][/size]
Patients should be checked for the following medications before the urine test: diuretics, aspirin, pyrazinamide (Tebrazid), phenylbutazone, probenecid (Benemid), and allopurinol (Lopurin). If the patient needs to continue taking these medications, the laboratory should be notified. Ignorance is bliss, is it? Isn't it better to learn more than not to know about something like Uric Acid Kidneys. So we have produced this article so that you can learn more about it!
[size=large][b]Description[/b][/size][hr] The uric acid blood test is performed on a sample of the patient's blood, withdrawn from a vein into a vacuum tube. The procedure, which is called a venipuncture, takes about five minutes. The urine test requires the patient to collect all urine voided over a 24-hour period, with the exception of the very first specimen. The patient keeps the specimen container on ice or in the refrigerator during the collection period.
[size=large][b]The Internet Guide: Gout[/b][/size][hr]Http://gout.smartadsscrets.com The Internet Guide: Purines - http://gout.smartadssecrets.com/purines A rolling stone gathers no moss. So if I just go on writing, and you don't understand, then it is of no use of me writing about Kidney Stone! Whatever written should be understandable by the reader.
[size=medium][b]Urine test[/b][/size]
Reference values for 24-hour urinary uric acid vary from laboratory to laboratory but are generally found within the following range: 250-750 mg/24 hours. It is with much interest that we got about to write on Allopurinol. So we do hope that you too read this article with the same, if not more interest!
[size=large][b]Abnormally Low Uric Acid Levels May Indicate that the[/b][/size][hr]Patient is taking allopurinol or probenecid for treatment of gout; may be pregnant; or suffers from Wilson's disease or Fanconi's syndrome. Writing is something that has to be done when one is in the mood to write. So when we got in the mood to write about Uric Acid Gout, nothing could stop us from writing!
[size=large][b]Abnormal results[/b][/size][hr] The critical value for the blood test is a level of uric acid higher than 12 milligrams per deciliter (about 4 ounces). We were actually wondering how to get about to writing about Purine Metabolism. However once we started writing, the words just seemed to flow continuously!
.
[size=large][b]Precautions[/b][/size][hr]
[size=medium][b]Blood test[/b][/size]
Patients scheduled for a blood test for uric acid should be checked for the following medications: loop diuretics (Diamox, Bumex, Edecrin, or Lasix); ethambutol (Myambutol); vincristine (Oncovin); pyrazinamide (Tebrazid); thiazide diuretics (Naturetin, Hydrex, Diuril, Esidrix, HydroDiuril, Aquatensen, Renese, Diurese); aspirin (low doses); acetaminophen (Tylenol); ascorbic acid (vitamin C preparations); levodopa (Larodopa); or phenacetin. These drugs can affect test results. It is rather inviting to go on writing on Kidney Stones. however as there is a limitation to the number of words to be written, we have confined ourselves to this. However, do enjoy yourself reading it.
[size=medium][b]Key Terms[/b][/size]
<dl> <dl> <dt>[b]Fanconi's syndrome[/b]</dt> <dd>A rare disorder caused by vitamin D deficiency or exposure to heavy metals. </dd> <dt>[b]Gout[/b]</dt> <dd>A metabolic disorder characterized by sudden recurring attacks of arthritis caused by deposits of crystals that build up in the joints due to abnormally high uric acid blood levels. In gout, uric acid may be overproduced, underexcreted, or both. </dd> <dt>[b]Hyperuricemia[/b]</dt> <dd>Excessively high levels of uric acid in the blood, often producing gout. </dd> <dt>[b]Purine[/b]</dt> <dd>A white crystalline substance that is one of the building blocks of DNA. Uric acid is produced when purine is broken down in the body. </dd> <dt>[b]Uric acid[/b]</dt> <dd>A compound resulting from the body's breakdown of purine. It is normally present in human urine only in small amounts. </dd> <dt>[b]Uricosuria[/b]</dt> <dd>Increased levels of uric acid in the urine. </dd> <dt>[b]Wilson's disease[/b]</dt> <dd>A rare hereditary disease marked by the buildup of copper in the liver and brain, causing loss of kidney function.</dd> </dl> </dl>
[size=medium][b]Further Reading[/b][/size]
[size=medium][b]For Your Information[/b][/size]
[list]
[size=medium][b]Books[/b][/size]
[*][i]Laboratory Test Handbook,[/i] edited by David S. Jacobs. Cleveland, OH: Lexi-Comp Inc., 1996. [*][i]Mosby's Diagnostic and Laboratory Test Reference,[/i] edited by Kathleen Deska Pagana and Timothy James Pagana. St. Louis: Mosby-Year Book, Inc., 1998. [*]Springhouse Corporation. [i]Handbook of Diagnostic Tests,[/i] edited by Matthew Cahill. Springhouse, PA: Springhouse Corporation, 1995. [/list]
[size=medium][b]Bio[/b][/size]
Do not judge a book by its cover; so don't just scan through this matter on Uric Acid Blood Test. read it thoroughly to judge its value and importance.
[size=large][b]Steve Warshaw[/b][/size][hr]Certified Personal Trainer and Nutritionist With over 15 years expereience developing training and nutrition programs for top level executives from companies such as Microsoft, Boeing, and Symetra Corp, Steve has established himself as a health and wellness expert.
[size=large][b]You Wish to Learn More about Gout, Purines, or Uric Acid,[/b][/size][hr]Check out Steve's s The Internet Guides. The Internet Guide: Uric Acid: http://gout.smartadssecrets.com/uric_acid It is rather interesting to note that people like reading about Uric Acid Blood Levels if they are presented in an easy and clear way. The presentation of an article too is important for one to entice people to read it!
[size=large][b]Certain Foods that are High in Purine May Increase the[/b][/size][hr]Patient's levels of uric acid. These include kidneys, liver, sweetbreads, sardines, anchovies, and meat extracts. Isn't it amazing how much information can be transferred through a single page? So much stands to gain, and to lose about Kidney Stones through a single page.
Increased [i]production[/i] of uric acid may result from eating foods that are high in purine. Increased uric acid levels due to overproduction may also be caused by gout, by a genetic disorder of purine metabolism, or by metastatic cancer, destruction of red blood cells, leukemia, or cancer chemotherapy. It is always better to have compositions with as little corrections in it as possible. This is why we have written this composition on Uric Acid Test with no corrections for the reader to be more interested in reading it.
[list][*]The laboratory should also be notified if the patient has had recent x-ray tests requiring contrast dyes.[*]These chemicals increase uric acid levels in urine and decrease them in blood. [/list]
[size=large][b]Normal results[/b][/size][hr]
[size=medium][b]Blood test[/b][/size]
Reference values for blood uric acid vary from laboratory to laboratory but are generally found within the following range: Male: 2.1-8.5 mg/dL; female: 2.0-6.6 mg/dL. Values may be slightly higher in the elderly. As the information we produce in our writing on Allopurinol may be utilized by the reader for informative purposes, it is very important that the information we provide be true. We have indeed maintained this.
[list][*]Decreased [i]excretion[/i] of uric acid is seen in chronic kidney disease, low thyroid, toxemia of pregnancy, and alcoholism.[*]Patients with gout excrete less than half the uric acid in their blood as other persons.[*]Only 10-15% of the total cases of hyperuricemia, however, are caused by gout.[/list]
Arthritis is a disease that causes pain and loss of movement of the joints. Joint pain is referred to as arthralgia. Arthritis is the leading cause of disability in people over the age of 55. The causes of arthritis depend on the form of arthritis. Causes include injury (leading to osteoarthritis), abnormal metabolism (such as gout and pseudogout), inheritance, infections, and for unclear reasons (such as rheumatoid arthritis and systemic lupus erythematosus). There are many forms of arthritis .There are about 200 different kinds of arthritis. The most common type is osteoarthritis (or degenerative arthritis), where the cartilage that protects the bones gets worn away. This makes joints stiff, painful and creaky. About 5 million people in the UK have osteoarthritis. OA is a chronic degenerative arthropathy that frequently leads to chronic pain and disability. With the aging of our population, this condition is becoming increasing prevalent and its treatment increasingly financially burdensome. Using radiographic criteria, the distal and proximal interphalangeal joints of the hand have been identified as the joints most commonly affected by OA, but they are the least likely to be symptomatic. Age is the most consistently identified risk factor for OA, regardless of the joint being studied. Prevalence rates for both radiographic OA and, to a lesser extent, symptomatic OA rise steeply after age 50 in men and age 40 in women. Occupation-related repetitive injury and physical trauma contribute to the development of secondary (non-idiopathic) OA, sometimes occurring in joints that are not affected by primary (idiopathic) OA, such as the metacarpophalangeal joints, wrists and ankles.
[size=large][b]Septic Arthritis[/b][/size][hr]Also known is Pyogenic arthritis. Septic arthritis is infection, usually bacterial, in the joint cavity. Septic arthritis usually affects just one joint, though occasionally it may occur in more than one joint at a time. It is the most dangerous form of acute arthritis. The joint cavity is usually a sterile space, with synovial fluid and cellular matter including a few white blood cells. Many different types of bacteria (germs) can cause septic arthritis. Infection with a bacterium called Staph. aureus is the most common cause. Septic arthritis is inflammation of a synovial membrane with purulent effusion into the joint capsule, usually due to bacterial infection. This disease entity also is referred to in the literature as bacterial, suppurative, purulent, or infectious arthritis. The most common bacterial isolates in native joints include gram-positive cocci, with S. aureus found in 40% to 50% of the cases. Septic arthritis is uncommon from age 3 to adolescence. Children with septic arthritis are more likely than adults to be infected with group B streptococcus and Haemophilus influenza. Young children and older adults are most likely to develop septic arthritis. As the population ages, doctors are finding that septic arthritis is becoming more common. Symptoms of septic arthritis occur suddenly and are characterized by severe pain, swelling in the affected joint along with acute pain. Chills and fever are also common symptoms. Chronic septic arthritis (which occurs less frequently) is caused by organisms such as Mycobacterium tuberculosis and Candida albicans. The knee and the hip are the most commonly infected joints. Suppressing our knowledge on Attack Gout is not our intention here. In fact, we mean to let everyone know more about Attack Gout after reading this!
[size=large][b]Gout is One of the Most Painful Types of Arthritis[/b][/size][hr]Gout was once incorrectly thought to be a disease of the rich and famous, caused by consuming too much rich food and fine wine. Gout is a disease due to a congenital disorder of uric acid metabolism. Uric acid is produced when purines are broken down by enzymes in the liver. Purines can be generated by the body itself (via the breakdown of cells in normal cellular turnover) or can be ingested in purine-rich foods (e.g. seafood, beer). Gout usually attacks the big toe (approximately 75% of first attacks), however it can also affect other joints such as the ankle, heel, instep, knee, wrist, elbow, fingers, and spine. In some cases the condition may appear in the joints of the small toes which have become immobile due to impact injury earlier in life, causing poor blood circulation that leads to gout. Chronic gout can lead to deposits of hard lumps of uric acid in and around the joints, decreased kidney function, and kidney stones. An acute attack of gout is a highly inflammatory arthritis often with intense swelling, redness and warmth surrounding the joint. The inflammatory component is so intense, an acute attack of gout is often mistaken for a bacterial cellulitis. Gout is mainly treated with anti-inflammatory drugs. Corticosteroids (also called steroids), may be prescribed for people who cannot take NSAIDs. Steroids also work by decreasing inflammation. Steroids can be injected into the affected joint or given as pills. Colchicine is often used to treat gout and usually begins working within a few hours of taking it.
[size=medium][b]Uric Acid Symptoms: Learn What Causes Gout[/b][/size]
Rheumatoid arthritis (RA) is traditionally considered a chronic, inflammatory autoimmune disorder. Rheumatoid arthritis occurs when the body's defence mechanisms go into action when there's no threat and start attacking the joints and sometimes other parts of the body. RA affects 1 million Americans, or about 1% of the adult population in the United States. This disease is 2 to 3 times more common in women than in men, and generally affects people between the ages of 20 and However, young children can develop a form of RA called juvenile rheumatoid arthritis. Two of the 100 types of arthritis are rheumatoid arthritis and lupus. There are specific symptoms, distinguishing characteristics, as well as overlapping symptoms associated with rheumatoid arthritis and lupus. Rheumatoid arthritis is an additive polyarthritis, with the sequential addition of involved joints, in contrast to the migratory or evanescent arthritis of systemic lupus erythematosus or the episodic arthritis of gout. Occasionally, patients experience an explosive polyarticular onset occurring over 24 to 48 hours. Morning stiffness, persisting more than one hour but often lasting several hours, may be a feature of any inflammatory arthritis but is especially characteristic of rheumatoid arthritis. Its duration is a useful gauge of the inflammatory activity of the disease. This is a dependable source of information on Uric Acid Metabolism. All that has to be done to verify its authenticity is to read it!
[size=large][b]Psoriatic Arthritis is Related to the Skin Condition Psoriasis[/b][/size][hr]It occurs more commonly in patients with tissue type HLA-B There are five clinical patterns of psoriatic arthritis. First is Asymmetrical mono- and oligoarticular arthritis (30-50% of cases) is the most common presentation of psoriatic arthritis. Second is symmetrical polyarticular arthritis (30-50% of cases) is ultimately the most common form of psoriatic arthritis. Third is distal interphalangeal (DIP) joint involvement (25% of cases) is nearly always associated with nail manifestationsm. Fourth is Arthritis mutilans is affects less than 5% of patients and is a severe, deforming and destructive arthritis. This condition can progress over months or years causing severe joint damage. Fifth is Axial arthritis (30-35% of cases) may be different in character from ankylosing spondylitis, the prototypical HLA-B27-associated spondyloarthropathy. It may present as sacro-iliitis, which may be asymmetrical and asymptomatic, or spondylitis, which may occur without sacro-iliitis and may affect any level of the spine in "skip" fashion. Genetic factors appear to play an important role. There is a 70% concordance for psoriasis in monozygotic twins. There is a 50-fold increased risk of developing psoriatic arthritis in first-degree relatives of patients with the disease. Environmental factors have been implicated. Streptococcal infection can precipitate the development of guttate psoriasis. HIV infection can present with both psoriasis and psoriatic arthritis, as well as worsen existing disease. We have avoided adding flimsy points on Arthritis Gout, as we find that the addition of such points have no effect on Arthritis Gout.
[size=large][b]Definition[/b][/size][hr] Uric acid tests are tests that are done to measure the levels of uric acid in blood serum or in urine.
[size=large][b]Purpose[/b][/size][hr]
[size=medium][b]Urine test[/b][/size]
Patients should be checked for the following medications before the urine test: diuretics, aspirin, pyrazinamide (Tebrazid), phenylbutazone, probenecid (Benemid), and allopurinol (Lopurin). If the patient needs to continue taking these medications, the laboratory should be notified. Ignorance is bliss, is it? Isn't it better to learn more than not to know about something like Uric Acid Kidneys. So we have produced this article so that you can learn more about it!
[size=large][b]Description[/b][/size][hr] The uric acid blood test is performed on a sample of the patient's blood, withdrawn from a vein into a vacuum tube. The procedure, which is called a venipuncture, takes about five minutes. The urine test requires the patient to collect all urine voided over a 24-hour period, with the exception of the very first specimen. The patient keeps the specimen container on ice or in the refrigerator during the collection period.
[size=large][b]The Internet Guide: Gout[/b][/size][hr]Http://gout.smartadsscrets.com The Internet Guide: Purines - http://gout.smartadssecrets.com/purines A rolling stone gathers no moss. So if I just go on writing, and you don't understand, then it is of no use of me writing about Kidney Stone! Whatever written should be understandable by the reader.
[size=medium][b]Urine test[/b][/size]
Reference values for 24-hour urinary uric acid vary from laboratory to laboratory but are generally found within the following range: 250-750 mg/24 hours. It is with much interest that we got about to write on Allopurinol. So we do hope that you too read this article with the same, if not more interest!
[size=large][b]Abnormally Low Uric Acid Levels May Indicate that the[/b][/size][hr]Patient is taking allopurinol or probenecid for treatment of gout; may be pregnant; or suffers from Wilson's disease or Fanconi's syndrome. Writing is something that has to be done when one is in the mood to write. So when we got in the mood to write about Uric Acid Gout, nothing could stop us from writing!
[size=large][b]Abnormal results[/b][/size][hr] The critical value for the blood test is a level of uric acid higher than 12 milligrams per deciliter (about 4 ounces). We were actually wondering how to get about to writing about Purine Metabolism. However once we started writing, the words just seemed to flow continuously!
.[size=large][b]Precautions[/b][/size][hr]
[size=medium][b]Blood test[/b][/size]
Patients scheduled for a blood test for uric acid should be checked for the following medications: loop diuretics (Diamox, Bumex, Edecrin, or Lasix); ethambutol (Myambutol); vincristine (Oncovin); pyrazinamide (Tebrazid); thiazide diuretics (Naturetin, Hydrex, Diuril, Esidrix, HydroDiuril, Aquatensen, Renese, Diurese); aspirin (low doses); acetaminophen (Tylenol); ascorbic acid (vitamin C preparations); levodopa (Larodopa); or phenacetin. These drugs can affect test results. It is rather inviting to go on writing on Kidney Stones. however as there is a limitation to the number of words to be written, we have confined ourselves to this. However, do enjoy yourself reading it.
[size=medium][b]Key Terms[/b][/size]
<dl> <dl> <dt>[b]Fanconi's syndrome[/b]</dt> <dd>A rare disorder caused by vitamin D deficiency or exposure to heavy metals. </dd> <dt>[b]Gout[/b]</dt> <dd>A metabolic disorder characterized by sudden recurring attacks of arthritis caused by deposits of crystals that build up in the joints due to abnormally high uric acid blood levels. In gout, uric acid may be overproduced, underexcreted, or both. </dd> <dt>[b]Hyperuricemia[/b]</dt> <dd>Excessively high levels of uric acid in the blood, often producing gout. </dd> <dt>[b]Purine[/b]</dt> <dd>A white crystalline substance that is one of the building blocks of DNA. Uric acid is produced when purine is broken down in the body. </dd> <dt>[b]Uric acid[/b]</dt> <dd>A compound resulting from the body's breakdown of purine. It is normally present in human urine only in small amounts. </dd> <dt>[b]Uricosuria[/b]</dt> <dd>Increased levels of uric acid in the urine. </dd> <dt>[b]Wilson's disease[/b]</dt> <dd>A rare hereditary disease marked by the buildup of copper in the liver and brain, causing loss of kidney function.</dd> </dl> </dl>
[size=medium][b]Further Reading[/b][/size]
[size=medium][b]For Your Information[/b][/size]
[list]
[size=medium][b]Books[/b][/size]
[*][i]Laboratory Test Handbook,[/i] edited by David S. Jacobs. Cleveland, OH: Lexi-Comp Inc., 1996. [*][i]Mosby's Diagnostic and Laboratory Test Reference,[/i] edited by Kathleen Deska Pagana and Timothy James Pagana. St. Louis: Mosby-Year Book, Inc., 1998. [*]Springhouse Corporation. [i]Handbook of Diagnostic Tests,[/i] edited by Matthew Cahill. Springhouse, PA: Springhouse Corporation, 1995. [/list]
[size=medium][b]Bio[/b][/size]
Do not judge a book by its cover; so don't just scan through this matter on Uric Acid Blood Test. read it thoroughly to judge its value and importance.
[size=large][b]Steve Warshaw[/b][/size][hr]Certified Personal Trainer and Nutritionist With over 15 years expereience developing training and nutrition programs for top level executives from companies such as Microsoft, Boeing, and Symetra Corp, Steve has established himself as a health and wellness expert.
[size=large][b]You Wish to Learn More about Gout, Purines, or Uric Acid,[/b][/size][hr]Check out Steve's s The Internet Guides. The Internet Guide: Uric Acid: http://gout.smartadssecrets.com/uric_acid It is rather interesting to note that people like reading about Uric Acid Blood Levels if they are presented in an easy and clear way. The presentation of an article too is important for one to entice people to read it!
Quote:You wish to learn more about Steve or to request training or nutrition information, please check out his website: http://www.bodybuiltbyvictoria.com.
[size=large][b]Certain Foods that are High in Purine May Increase the[/b][/size][hr]Patient's levels of uric acid. These include kidneys, liver, sweetbreads, sardines, anchovies, and meat extracts. Isn't it amazing how much information can be transferred through a single page? So much stands to gain, and to lose about Kidney Stones through a single page.
Increased [i]production[/i] of uric acid may result from eating foods that are high in purine. Increased uric acid levels due to overproduction may also be caused by gout, by a genetic disorder of purine metabolism, or by metastatic cancer, destruction of red blood cells, leukemia, or cancer chemotherapy. It is always better to have compositions with as little corrections in it as possible. This is why we have written this composition on Uric Acid Test with no corrections for the reader to be more interested in reading it.
[list][*]The laboratory should also be notified if the patient has had recent x-ray tests requiring contrast dyes.[*]These chemicals increase uric acid levels in urine and decrease them in blood. [/list]
[size=large][b]Normal results[/b][/size][hr]
[size=medium][b]Blood test[/b][/size]
Reference values for blood uric acid vary from laboratory to laboratory but are generally found within the following range: Male: 2.1-8.5 mg/dL; female: 2.0-6.6 mg/dL. Values may be slightly higher in the elderly. As the information we produce in our writing on Allopurinol may be utilized by the reader for informative purposes, it is very important that the information we provide be true. We have indeed maintained this.
[list][*]Decreased [i]excretion[/i] of uric acid is seen in chronic kidney disease, low thyroid, toxemia of pregnancy, and alcoholism.[*]Patients with gout excrete less than half the uric acid in their blood as other persons.[*]Only 10-15% of the total cases of hyperuricemia, however, are caused by gout.[/list]
Arthritis is a disease that causes pain and loss of movement of the joints. Joint pain is referred to as arthralgia. Arthritis is the leading cause of disability in people over the age of 55. The causes of arthritis depend on the form of arthritis. Causes include injury (leading to osteoarthritis), abnormal metabolism (such as gout and pseudogout), inheritance, infections, and for unclear reasons (such as rheumatoid arthritis and systemic lupus erythematosus). There are many forms of arthritis .There are about 200 different kinds of arthritis. The most common type is osteoarthritis (or degenerative arthritis), where the cartilage that protects the bones gets worn away. This makes joints stiff, painful and creaky. About 5 million people in the UK have osteoarthritis. OA is a chronic degenerative arthropathy that frequently leads to chronic pain and disability. With the aging of our population, this condition is becoming increasing prevalent and its treatment increasingly financially burdensome. Using radiographic criteria, the distal and proximal interphalangeal joints of the hand have been identified as the joints most commonly affected by OA, but they are the least likely to be symptomatic. Age is the most consistently identified risk factor for OA, regardless of the joint being studied. Prevalence rates for both radiographic OA and, to a lesser extent, symptomatic OA rise steeply after age 50 in men and age 40 in women. Occupation-related repetitive injury and physical trauma contribute to the development of secondary (non-idiopathic) OA, sometimes occurring in joints that are not affected by primary (idiopathic) OA, such as the metacarpophalangeal joints, wrists and ankles.
[size=large][b]Septic Arthritis[/b][/size][hr]Also known is Pyogenic arthritis. Septic arthritis is infection, usually bacterial, in the joint cavity. Septic arthritis usually affects just one joint, though occasionally it may occur in more than one joint at a time. It is the most dangerous form of acute arthritis. The joint cavity is usually a sterile space, with synovial fluid and cellular matter including a few white blood cells. Many different types of bacteria (germs) can cause septic arthritis. Infection with a bacterium called Staph. aureus is the most common cause. Septic arthritis is inflammation of a synovial membrane with purulent effusion into the joint capsule, usually due to bacterial infection. This disease entity also is referred to in the literature as bacterial, suppurative, purulent, or infectious arthritis. The most common bacterial isolates in native joints include gram-positive cocci, with S. aureus found in 40% to 50% of the cases. Septic arthritis is uncommon from age 3 to adolescence. Children with septic arthritis are more likely than adults to be infected with group B streptococcus and Haemophilus influenza. Young children and older adults are most likely to develop septic arthritis. As the population ages, doctors are finding that septic arthritis is becoming more common. Symptoms of septic arthritis occur suddenly and are characterized by severe pain, swelling in the affected joint along with acute pain. Chills and fever are also common symptoms. Chronic septic arthritis (which occurs less frequently) is caused by organisms such as Mycobacterium tuberculosis and Candida albicans. The knee and the hip are the most commonly infected joints. Suppressing our knowledge on Attack Gout is not our intention here. In fact, we mean to let everyone know more about Attack Gout after reading this!
[size=large][b]Gout is One of the Most Painful Types of Arthritis[/b][/size][hr]Gout was once incorrectly thought to be a disease of the rich and famous, caused by consuming too much rich food and fine wine. Gout is a disease due to a congenital disorder of uric acid metabolism. Uric acid is produced when purines are broken down by enzymes in the liver. Purines can be generated by the body itself (via the breakdown of cells in normal cellular turnover) or can be ingested in purine-rich foods (e.g. seafood, beer). Gout usually attacks the big toe (approximately 75% of first attacks), however it can also affect other joints such as the ankle, heel, instep, knee, wrist, elbow, fingers, and spine. In some cases the condition may appear in the joints of the small toes which have become immobile due to impact injury earlier in life, causing poor blood circulation that leads to gout. Chronic gout can lead to deposits of hard lumps of uric acid in and around the joints, decreased kidney function, and kidney stones. An acute attack of gout is a highly inflammatory arthritis often with intense swelling, redness and warmth surrounding the joint. The inflammatory component is so intense, an acute attack of gout is often mistaken for a bacterial cellulitis. Gout is mainly treated with anti-inflammatory drugs. Corticosteroids (also called steroids), may be prescribed for people who cannot take NSAIDs. Steroids also work by decreasing inflammation. Steroids can be injected into the affected joint or given as pills. Colchicine is often used to treat gout and usually begins working within a few hours of taking it.
[size=medium][b]Uric Acid Symptoms: Learn What Causes Gout[/b][/size]
Rheumatoid arthritis (RA) is traditionally considered a chronic, inflammatory autoimmune disorder. Rheumatoid arthritis occurs when the body's defence mechanisms go into action when there's no threat and start attacking the joints and sometimes other parts of the body. RA affects 1 million Americans, or about 1% of the adult population in the United States. This disease is 2 to 3 times more common in women than in men, and generally affects people between the ages of 20 and However, young children can develop a form of RA called juvenile rheumatoid arthritis. Two of the 100 types of arthritis are rheumatoid arthritis and lupus. There are specific symptoms, distinguishing characteristics, as well as overlapping symptoms associated with rheumatoid arthritis and lupus. Rheumatoid arthritis is an additive polyarthritis, with the sequential addition of involved joints, in contrast to the migratory or evanescent arthritis of systemic lupus erythematosus or the episodic arthritis of gout. Occasionally, patients experience an explosive polyarticular onset occurring over 24 to 48 hours. Morning stiffness, persisting more than one hour but often lasting several hours, may be a feature of any inflammatory arthritis but is especially characteristic of rheumatoid arthritis. Its duration is a useful gauge of the inflammatory activity of the disease. This is a dependable source of information on Uric Acid Metabolism. All that has to be done to verify its authenticity is to read it!
[size=large][b]Psoriatic Arthritis is Related to the Skin Condition Psoriasis[/b][/size][hr]It occurs more commonly in patients with tissue type HLA-B There are five clinical patterns of psoriatic arthritis. First is Asymmetrical mono- and oligoarticular arthritis (30-50% of cases) is the most common presentation of psoriatic arthritis. Second is symmetrical polyarticular arthritis (30-50% of cases) is ultimately the most common form of psoriatic arthritis. Third is distal interphalangeal (DIP) joint involvement (25% of cases) is nearly always associated with nail manifestationsm. Fourth is Arthritis mutilans is affects less than 5% of patients and is a severe, deforming and destructive arthritis. This condition can progress over months or years causing severe joint damage. Fifth is Axial arthritis (30-35% of cases) may be different in character from ankylosing spondylitis, the prototypical HLA-B27-associated spondyloarthropathy. It may present as sacro-iliitis, which may be asymmetrical and asymptomatic, or spondylitis, which may occur without sacro-iliitis and may affect any level of the spine in "skip" fashion. Genetic factors appear to play an important role. There is a 70% concordance for psoriasis in monozygotic twins. There is a 50-fold increased risk of developing psoriatic arthritis in first-degree relatives of patients with the disease. Environmental factors have been implicated. Streptococcal infection can precipitate the development of guttate psoriasis. HIV infection can present with both psoriasis and psoriatic arthritis, as well as worsen existing disease. We have avoided adding flimsy points on Arthritis Gout, as we find that the addition of such points have no effect on Arthritis Gout.